Analysis of embryonic fibroblast cells lacking Werner syndrome (WS) protein (Wrn), or poly(ADP-ribose) polymerase-1 (PARP-1), or both. Mutations in Wrn or PARP-1 result in genomic instability. Results provide insight into tumor progression, senescence, oxidative stress and aging.
- Organism:
- Mus musculus
- Type:
- Expression profiling by array,
log ratio,
3 genotype/variation sets
- Platform:
- GPL891
- Series:
- GSE3359
- 6 Samples
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