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Gene expression profiles of MV-4-11 AML cells treated HDAC1/2 -selective inhibitor and Azacitidine
PubMed Full text in PMC Similar studies Analyze with GEO2R
Effects of BRD4 inhibition in AML
PubMed Similar studies Analyze with GEO2R
Differential gene expression analysis of pediatric acute myeloid leukemia cell lines treated with azacytidine and panobinostat alone or in combination and compared to vehicle treated cells.
Chemical inhibition of Histone Deacetylases 1 and 2 induces fetal hemoglobin through activation of Gata2
ChIP-seq of erythroid progenitors treated with vehicle or the HDAC1/2-selective inhibitor ACY-957
PubMed Full text in PMC Similar studies SRA Run Selector
Gene expression profiles of erythroid progenitors treated with vehicle or the HDAC1/2-selective inhibitor ACY-957
Rational targeting of cooperating layers of the epigenome yields enhanced therapeutic efficacy against AML
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Effects of Beta Catenin antagonist BC2059 in AML cells
Azacitidine treated AML cells reveals highly variable cell surface proteome remodeling
Azacitidine treated AML cells reveals highly variable cell surface proteome remodeling [Affymetrix]
Azacitidine treated AML cells reveals highly variable cell surface proteome remodeling [Illumina]
Romidepsin and azacitidine synergize in their epigenetic modulatory effects to induce apoptosis in CTCL
PubMed Similar studies SRA Run Selector
OCI/AML2 ATRA AND VPA GENE EXPRESSION
Acute myeloblastic leukemia cells response to all trans retinoic acid and valproic acid
PubMed Similar studies GEO Profiles Analyze DataSet
Targeting miR-126 in inv(16) acute myeloid leukemia inhibits leukemia development and leukemia stem cell maintenance
State-Transition Analysis of Time-Sequential microRNA Expression Predicts Development of Acute Myeloid Leukemia
State-Transition Analysis of Time-Sequential Gene Expression Identifies Critical Points That Predict Leukemia Development
In vitro and in vivo monitoring of valproic acid effects on gene expression signatures in adult acute myeloid leukemia
Epigenetic therapy as a novel approach for GFI136N-associated AML
Effect of Gfi1 36N variant on genome-wide H3K9 Acetylation patterns
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