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Modulation of type-I interferon responses in Rhesus Macaque whole blood
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Modulation of type-I interferon responses reduces SARS-CoV-2 replication and inflammation in rhesus macaques
Short-term pIFN-α2a treatment does not significantly reduce the viral reservoir of SIV-infected, ART-treated rhesus macaques
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Longitudinal single-cell RNA-sequencing (scRNAseq) of broncho-alaveolar compartment of rhesus macaque model of COVID-19
Baricitinib treatment resolves lower airway inflammation and neutrophil recruitment in SARS-CoV-2-infected rhesus macaques
Baricitinib treatment resolves lower airway inflammation and neutrophil recruitment in SARS-CoV-2-infected rhesus macaques [RNA-Seq]
Baricitinib treatment resolves lower airway inflammation and neutrophil recruitment in SARS-CoV-2-infected rhesus macaques [scRNA-Seq]
Primary Human Airway Epithelial Cultures infected with SARS-CoV-2
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Reduced chronic lymphocyte activation following Interferon-α blockade in the acute phase of SIV infection in rhesus macaques
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CCR2-dependent monocyte-derived cells restrict SARS-CoV-2 infection
Cytokines and lipid mediators of inflammation in lungs of SARS-CoV-2 infected mice
Differential interferon-α immune signatures prevent SARS-CoV-2 infection
Type I interferon response in human HTBE and A549 cells
Mild SARS-CoV-2 infection in rhesus macaques is associated with viral control prior to antigen-specific T cell responses in tissues
Early differentially expressed immunological genes in mild and severe COVID-19
TREM2+ and interstitial macrophages orchestrate airway inflammation in SARS-CoV-2 infection in rhesus macaques
USP22 controls type III interferon signaling and SARS-CoV-2 infection through activation of STING
Dynamic Innate Immune Responses of Human Bronchial Epithelial Cells against SARS-CoV and DOHV infection
SM001: SARS CoV MA15 infection of C57Bl/6 mouse model – Data from 4 viral doses at 1, 2, 4 and 7 days post infection.
Pharmacological Activation of STING blocks SARS-CoV-2 infection
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